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1. 15 DISRUPTION OF FOXP3-EZH2 INTERACTION BY GENETIC MUTATION OR SIGNALING-INDUCED INACTIVATION REPRESENTS A NOVEL PATHOBIOLOGICAL MECHANISM IN INFLAMMATORY BOWEL DISEASE. (18th January 2018)

2. Computational modeling reveals key molecular properties and dynamic behavior of disruptor of telomeric silencing 1‐like (DOT1L) and partnering complexes involved in leukemogenesis. Issue 1 (12th September 2021)

3. Diabetes‐Causing Gene, Kruppel‐Like Factor 11, Modulates the Antinociceptive Response of Chronic Ethanol Intake. (15th January 2014)

4. Functional Characterization of Nupr1L, A Novel p53‐Regulated Isoform of the High‐Mobility Group (HMG)‐Related Protumoral Protein Nupr1. Issue 12 (24th August 2015)

5. Gene expression profiling of patient‐derived pancreatic cancer xenografts predicts sensitivity to the BET bromodomain inhibitor JQ1: implications for individualized medicine efforts. Issue 4 (8th March 2017)

6. Genetic inactivation of Nupr1 acts as a dominant suppressor event in a two-hit model of pancreatic carcinogenesis. Issue 6 (11th September 2013)

7. Homotypic cell cannibalism, a cell‐death process regulated by the nuclear protein 1, opposes to metastasis in pancreatic cancer. Issue 5 (7th May 2021)

8. Inactivation of NUPR1 promotes cell death by coupling ER-stress responses with necrosis. Issue 1 (December 2018)

10. Novel role of VMP1 as modifier of the pancreatic tumor cell response to chemotherapeutic drugs1. Issue 9 (25th May 2013)