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1. Myostatin inhibition prevents skeletal muscle pathophysiology in Huntington's disease mice. Issue 1 (December 2017)

2. Phenotype onset in Huntington's disease knock‐in mice is correlated with the incomplete splicing of the mutant huntingtin gene. Issue 12 (7th July 2019)

3. B3 Comparison of the effect of a pure CAG repeat and mixed cagcaa repeat on the extent to which the htt gene is aberrantly spliced in knock-in mice. (13th September 2016)

4. B4 Detection of the aberrantly spliced exon 1 – intron 1 htt mRNA in HD patient post mortem brain tissue and fibroblast lines. (13th September 2016)

6. B1 HTT CAG knock-in mice with pure and interrupted repeat tracts provide insight into the role of somatic expansion in HD pathogenesis. (13th September 2016)

7. Development of novel bioassays to detect soluble and aggregated Huntingtin proteins on three technology platforms. Issue 1 (5th January 2021)

8. B03 Analysis of a Huntington's disease knock-in mouse model designed to prevent the generation of the exon 1 HTT protein. (12th September 2022)

9. Mutations causing Lopes-Maciel-Rodan syndrome are huntingtin hypomorphs. Issue 3 (11th January 2021)

10. High resolution time-course mapping of early transcriptomic, molecular and cellular phenotypes in Huntington's disease CAG knock-in mice across multiple genetic backgrounds. (27th February 2017)