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2. CaMKIIδ subtypes differentially regulate infarct formation following ex vivo myocardial ischemia/reperfusion through NF-κB and TNF-α. (February 2017)

4. Hrd1 and ER-Associated Protein Degradation, ERAD, Are Critical Elements of the Adaptive ER Stress Response in Cardiac Myocytes. Issue 6 (28th August 2015)

5. Junctophilin-2 gene therapy rescues heart failure by normalizing RyR2-mediated Ca2 + release. (15th December 2016)

6. Junctophilin-2 gene therapy rescues heart failure by normalizing RyR2-mediated Ca2 + release. (15th December 2016)

7. MANF, a structurally unique redox-sensitive chaperone, restores ER-protein folding in the ischemic heart. (November 2017)

10. Proteomic analysis of the cardiac myocyte secretome reveals extracellular protective functions for the ER stress response. (June 2020)