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1. Aberrant HRAS transcript processing underlies a distinctive phenotype within the RASopathy clinical spectrum. Issue 7 (3rd May 2017)

2. Biallelic SQSTM1 mutations in early-onset, variably progressive neurodegeneration. (24th July 2018)

3. Bone tissue homeostasis and risk of fractures in Costello syndrome: A 4‐year follow‐up study. Issue 2 (15th December 2021)

4. Characterization of bone homeostasis in individuals affected by cardio‐facio‐cutaneous syndrome. Issue 2 (2nd December 2021)

5. Compound heterozygosity for PTPN11 variants in a subject with Noonan syndrome provides insights into the mechanism of SHP2‐related disorders. Issue 3 (4th January 2021)

6. Cover Image, Volume 38, Issue 4. Issue 4 (April 2017)

7. Dominantly acting KIF5B variants with pleiotropic cellular consequences cause variable clinical phenotypes. Issue 3 (26th August 2022)

8. Hyperactive HRAS dysregulates energetic metabolism in fibroblasts from patients with Costello syndrome via enhanced production of reactive oxidizing species. Issue 4 (11th September 2021)

9. Loss of function of the E3 ubiquitin-protein ligase UBE3B causes Kaufman oculocerebrofacial syndrome. Issue 8 (17th May 2013)

10. Pathogenic PTPN11 variants involving the poly‐glutamine Gln255‐Gln256‐Gln257 stretch highlight the relevance of helix B in SHP2's functional regulation. Issue 6 (11th March 2020)