Impact of Septal Deviation on Recurrent Chronic Rhinosinusitis after Primary Surgery: A Matched Case‐Control Study. (27th November 2018)

• Record Type: Journal Article
• Authors: Fu, Terence; Lee, Daniel; Yip, Jonathan; Jamal, Alisha; Lee, John M.
• Is Part Of: Otolaryngology--head and neck surgery. Volume 160:Number 5(2019)
• Page Start: 922
• Page End: 927
• Publication Date: 2018-11-27
• Subjects: septal deviation -- rhinosinusitis -- endoscopic sinus surgery; Head -- Surgery -- Periodicals; Neck -- Surgery -- Periodicals; Otolaryngology -- Periodicals; 617.51
• DOI: 10.1177/0194599818815106
• Languages: English
• ISSNs: 0194-5998

Abstract

Abstract : Objective: To evaluate the impact of untreated deviated nasal septum (DNS) on recalcitrant chronic rhinosinusitis (CRS) among patients undergoing revision endoscopic sinus surgery (ESS). Study Design: Case‐control study. Setting: Tertiary academic center. Subjects and Methods: We performed a retrospective review of 489 patients undergoing revision ESS for CRS at a tertiary academic center. Patients undergoing septoplasty were matched to nonseptoplasty controls based on age and sex. Preoperative Lund‐Mackay score (LMS) was compared between cohorts. Linear regression was used to identify predictors of LMS and ostiomeatal complex (OMC) obstruction. Results: Thirty‐six matched pairs (72 patients) were selected for analysis: 36 undergoing septoplasty and revision ESS and 36 undergoing revision ESS alone. Compared with nonseptoplasty controls, the septoplasty group had a significantly higher average LMS (17.8 vs 14.6, P =. 02) and a greater rate of OMC obstruction (89% vs 61%, P <. 01). The septoplasty group also had significantly higher opacification scores in the maxillary (1.5 vs 1.2, P =. 03) and posterior ethmoid (1.8 vs 1.4, P =. 02) sinuses. On multivariable analysis, DNS was an independent predictor of LMS ( P =. 02) and OMC obstruction ( P <. 01). Conclusion: Untreated DNS is associated with radiographic markers of CRS severity among patients undergoing revision ESS and may contribute to the multifactorial pathogenesis of persistent CRS.