Current recommendations and novel strategies for sustainable management of soybean sudden death syndrome. Issue 10 (29th May 2021)

• Record Type: Journal Article
• Authors: Rodriguez, Maria C; Sautua, Francisco; Scandiani, Mercedes; Carmona, Marcelo; Asurmendi, Sebastián
• Is Part Of: Pest management science. Volume 77:Issue 10(2021)
• Page Start: 4238
• Page End: 4248
• Publication Date: 2021-05-29
• Subjects: sudden death syndrome -- Fusarium virguliforme -- Fusarium tucumaniae -- disease control -- integrated disease management -- Glycine max; Pests -- Control -- Periodicals; Pesticides -- Periodicals; 632.9
• DOI: 10.1002/ps.6458
• Languages: English
• ISSNs: 1526-498X

Abstract

Abstract: The increase in food production requires reduction of the damage caused by plant pathogens, minimizing the environmental impact of management practices. Soil‐borne pathogens are among the most relevant pathogens that affect soybean crop yield. Soybean sudden death syndrome (SDS), caused by several distinct species of Fusarium, produces significant yield losses in the leading soybean‐producing countries in North and South America. Current management strategies for SDS are scarce since there are no highly resistant cultivars and only a few fungicide seed treatments are available. Because of this, innovative approaches for SDS management need to be developed. Here, we summarize recently explored strategies based on plant nutrition, biological control, priming of plant defenses, host‐induced gene silencing, and the development of new SDS‐resistance cultivars using precision breeding techniques. Finally, sustainable management of SDS should also consider cultural control practices with minimal environmental impact. © 2021 Society of Chemical Industry. Abstract : Novel strategies for soybean sudden death syndrome management based on chemical control, cultural control, resistant cultivars obtained through conventional breeding and new breeding techniques, plant nutrition, biological control, priming of plant defenses, and host‐induced gene silencing.