Are sensorimotor strokes lacunar strokes? A case-control study of lacunar and non-lacunar infarcts. Issue 12 (December 1991)

• Record Type: Journal Article
• Authors: Landi, G; Anzalone, N; Cella, E; Boccardi, E; Musicco, M
• Is Part Of: Journal of neurology, neurosurgery and psychiatry. Volume 54:Issue 12(1991)
• Page Start: 1063
• Page End: 1068
• Publication Date: 1991-12
• Subjects: Neurology -- Periodicals; Nervous system -- Surgery -- Periodicals; Psychiatry -- Periodicals; 616.8
• DOI: 10.1136/jnnp.54.12.1063
• Languages: English
• ISSNs: 0022-3050

Abstract

Abstract : To determine whether sensorimotor strokes should be considered as lacunar syndromes 34 consecutive patients with first-ever ischaemic sensorimotor stroke were evaluated and compared with 103 patients with non-lacunar infarcts and another 88 patients with lacunar infarcts. Potential thromboembolic sources were more frequent in patients with non-lacunar infarcts (p = 0.003, versus sensorimotor strokes). Although the overall prevalence of hypodense lesions at CT scan was not significantly different among the three groups, lacunar lesions were found in 47.1% of sensorimotor strokes, compared with 6.8% of non-lacunar infarcts (p less than 0.0001). In a mean follow up period of 28.7 months, the incidence of stroke and myocardial infarction among sensorimotor strokes was similar to that of patients with lacunar infarct, but significantly lower than in non-lacunar infarcts (p less than 0.05). These results demonstrate important differences between sensorimotor and non-lacunar infarcts, but quite similar findings in sensorimotor and lacunar strokes, and thus support the theory that sensorimotor strokes are commonly due to lacunar lesions.