Eagle Syndrome as a Cause of Cerebral Venous Sinus Thrombosis. (1st April 2019)

• Record Type: Journal Article
• Authors: Zhang, Fu-Liang; Zhou, Hong-Wei; Guo, Zhen-Ni; Yang, Yi
• Is Part Of: Canadian journal of neurological sciences. Volume 46:Number 3(2019)
• Page Start: 344
• Page End: 345
• Publication Date: 2019-04-01
• Subjects: Eagle syndrome, -- Cerebral venous sinus thrombosis; Neurology -- Periodicals; Nervous system -- Surgery -- Periodicals; Electronic journals; 616.8
• DOI: 10.1017/cjn.2019.17
• Languages: English
• ISSNs: 0317-1671

Abstract

Abstract : A 15-year-old teenager presented with a 2-month history of headache. Neurological examination was normal except for papilledema. Further lumbar puncture indicated intracranial hypertension (330 mm H2 O). Brain magnetic resonance imaging (MRI) was normal but phase contrast-magnetic resonance venography (PC-MRV) (Figure 1 (A)) suggested possible left transverse-sigmoid sinus thrombosis; subsequent contrast-enhanced 3D fat-saturated T1 volumetric isotropic turbo spin echo acquisition (VISTA) MRI (Figure 1(B)) confirmed the pathology. Hyper-coagulable panel results (including six steroid sex hormones, antithrombin III, protein C, protein S, lupus anticoagulant, and anticardiolipin antibodies) were all within normal range. In further examination, computed tomography (CT) venography images (Figure 1(C) and (D)) showed that the left jugular vein was compressed by the styloid process, consistent with Eagle syndrome. 1 The patient who refused the recommended surgical treatment, however, chose anticoagulant therapy consisting of low-molecular weight heparin subcutaneous injection in addition to new oral anticoagulant. At 18-month follow-up, the patient reported no symptoms remained.