Liver-chip identifies mitochondrial dysfunction, oxidative stress, and innate immune response as potential pathways of toxicity for the GPR40 agonist TAK-875. (10th October 2018) - Jang, K.-J.; Otieno, M.; Ronxhi, J.; Kodella, K.; Rubins, J.; Simic, D.; Singer, M.; Lim, H.-K.; Lam, W.; Chen, J.; Petropolis, D.; Kulkarni, G.; et al.