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Impaired homeostasis of hypoxia-derived reactive oxygen species during endochondral ossification repair defines a molecular mechanism for fracture nonunion. (April 2016)
Record Type:
Journal Article
Title:
Impaired homeostasis of hypoxia-derived reactive oxygen species during endochondral ossification repair defines a molecular mechanism for fracture nonunion. (April 2016)
Main Title:
Impaired homeostasis of hypoxia-derived reactive oxygen species during endochondral ossification repair defines a molecular mechanism for fracture nonunion