Cite
HARVARD Citation
Lebeau, P. et al. (2018). Loss-of-function PCSK9 Mutants Evade the Unfolded Protein Response Sensor, GRP78, and Fail to Induce Endoplasmic Reticulum Stress when Retained. Atherosclerosis. pp. 139-. [Online].
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Lebeau, P. et al. (2018). Loss-of-function PCSK9 Mutants Evade the Unfolded Protein Response Sensor, GRP78, and Fail to Induce Endoplasmic Reticulum Stress when Retained. Atherosclerosis. pp. 139-. [Online].