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Rivaroxaban inhibits oxidative and inflammatory reactions in advanced glycation end product-exposed tubular cells by blocking thrombin/protease-activated receptor-2 system. Issue 4 (April 2015)
Record Type:
Journal Article
Title:
Rivaroxaban inhibits oxidative and inflammatory reactions in advanced glycation end product-exposed tubular cells by blocking thrombin/protease-activated receptor-2 system. Issue 4 (April 2015)
Main Title:
Rivaroxaban inhibits oxidative and inflammatory reactions in advanced glycation end product-exposed tubular cells by blocking thrombin/protease-activated receptor-2 system
Graphical Abstract: Highlights: AGEs increased PAR-2 levels in tubular cells. Factor Xa increased ROS and MCP-1 mRNA levels in tubular cells. AGEs potentiated the effects of Xa on tubular cells. Rivaroxaban or an antagonist of PAR-2 blocked the harmful actions of Xa.