Finite element prediction of fatigue damage growth in cancellous bone. Issue 5 (3rd April 2016)

• Record Type: Journal Article
• Authors: Hambli, Ridha; Frikha, Sana; Toumi, Hechmi; Tavares, João Manuel R. S.
• Is Part Of: Computer methods in biomechanics and biomedical engineering. Volume 19:Issue 5(2016)
• Page Start: 563
• Page End: 570
• Publication Date: 2016-04-03
• Subjects: cancellous bone -- microdamage -- fatigue -- microcracks -- finite element; Biomechanics -- Data processing -- Periodicals; Biomedical engineering -- Periodicals; Biomechanics -- Periodicals; Biomedical Engineering -- methods -- Periodicals; Computing Methodologies -- Periodicals; 612.7
• DOI: 10.1080/10255842.2015.1048687
• Languages: English
• ISSNs: 1025-5842

Abstract

Abstract : Cyclic stresses applied to bones generate fatigue damage that affects the bone stiffness and its elastic modulus. This paper proposes a finite element model for the prediction of fatigue damage accumulation and failure in cancellous bone at continuum scale. The model is based on continuum damage mechanics and incorporates crack closure effects in compression. The propagation of the cracks is completely simulated throughout the damaged area. In this case, the stiffness of the broken element is reduced by 98% to ensure no stress-carrying capacities of completely damaged elements. Once a crack is initiated, the propagation direction is simulated by the propagation of the broken elements of the mesh. The proposed model suggests that damage evolves over a real physical time variable (cycles). In order to reduce the computation time, the integration of the damage growth rate is based on the cycle blocks approach. In this approach, the real number of cycles is reduced (divided) into equivalent blocks of cycles. Damage accumulation is computed over the cycle blocks and then extrapolated over the corresponding real cycles. The results show a clear difference between local tensile and compressive stresses on damage accumulation. Incorporating stiffness reduction also produces a redistribution of the peak stresses in the damaged region, which results in a delay in damage fracture.