Inflammation and nerve fiber interaction in endometriotic pain. (January 2015)

• Record Type: Journal Article
• Authors: McKinnon, Brett D.; Bertschi, Dominic; Bersinger, Nick A.; Mueller, Michael D.
• Is Part Of: Trends in endocrinology and metabolism. Volume 26:Number 1(2015)
• Page Start: 1
• Page End: 10
• Publication Date: 2015-01
• Subjects: Endocrinology -- Periodicals; Metabolism -- Periodicals; Metabolism; 616.4
• DOI: 10.1016/j.tem.2014.10.003
• Languages: English
• ISSNs: 1043-2760

Abstract

<abstract abstract-type="author-highlights" id="abs0005"> <title id="sect0005">Highlights</title> <sec> <p id="spar0005"> <list id="lis0005"> <list-item id="lsti0005"> <label></label> <p id="par0005">The pathogenesis of endometriotic pain is not clear.</p> </list-item> <list-item id="lsti0010"> <label></label> <p id="par0010">Endometriotic lesions are associated with inflammation and nerve growth.</p> </list-item> <list-item id="lsti0015"> <label></label> <p id="par0015">Production of chronic pain by endometriotic lesions can permanently affect the central nervous system.</p> </list-item> <list-item id="lsti0020"> <label></label> <p id="par0020">Endometriotic-associated inflammation may interact with the central nervous system to produce endometriotic pain.</p> </list-item> </list> </p> </sec> </abstract>