Gastrointestinal Hemodynamics in Dogs with Nonfood Induced Atopic Dermatitis. (3rd April 2013)

• Record Type: Journal Article
• Authors: Bruet, V.; Brune, J.; Pastor, A.; Imparato, L.; Roussel, A.; Bourdeau, P.; Desfontis, J.C.
• Is Part Of: Journal of veterinary internal medicine. Volume 27:Number 3(2013:May/Jun.)
• Page Start: 451
• Page End: 455
• Publication Date: 2013-04-03
• Subjects: Veterinary medicine -- Periodicals; 636.0896
• DOI: 10.1111/jvim.12072
• Languages: English
• ISSNs: 0891-6640

Abstract

<abstract abstract-type="main" xml:lang="en" id="jvim12072-abs-0001"> <title> <x xml:space="preserve">Abstract</x> </title> <sec id="jvim12072-sec-0001" sec-type="section"> <title>Background</title> <p>Canine atopic dermatitis can be a result of exposure to aeroallergens or trophallergens. Hemodynamic alterations occur in dogs with food hypersensitivity.</p> </sec> <sec id="jvim12072-sec-0002" sec-type="section"> <title>Hypothesis/Objectives</title> <p>To evaluate if hemodynamic alterations occur in dogs with NFICAD with lowered resistance to diastolic flow at fasting, after feeding, or both.</p> </sec> <sec id="jvim12072-sec-0003" sec-type="section"> <title>Animals</title> <p>Ten healthy dogs and 22 dogs with NFICAD were included from the hospital population.</p> </sec> <sec id="jvim12072-sec-0004" sec-type="section"> <title>Methods</title> <p>Blinded prospective study. Peak systolic velocity (PSV), end diastolic velocity (EDV), mean velocity (MV), pulsatility index (PI), resistive index (RI) and PSV/EDV ratio were measured at fasting for both arteries (cranial mesenteric artery [CMA], celiac artery [CA]) and at 40 minutes after feeding in CMA and at 60 minutes in CA. The results were analyzed statistically with a mixed model.</p> </sec> <sec id="jvim12072-sec-0005" sec-type="section"> <title>Results</title> <p>There was no difference detected between groups of dogs for any variable except EDV during fasting (<italic>P </italic>=<italic> </italic>.01).</p> </sec> <sec id="jvim12072-sec-0006" sec-type="section"> <title>Conclusions and Clinical Importance</title> <p>There is no decrease in resistance in NFICAD to diastolic flow. This observation could be explained by the absence intestinal inflammation in NFICAD.</p> </sec> </abstract>