Alcoholic/non-alcoholic digestive diseases. ([2019])

• Record Type: Book
• Further Information: Note: Hitoshi Yoshiji, Kosuke Kaji, editors.
• Editors: Yoshiji, Hitoshi; Kaji, Kosuke
• Contents: Intro; Preface; Contents; Part I: Alcoholic/Non-Alcoholic Gastrointestinal Diseases; Chapter 1: Alcohol-Induced DNA Injury in Esophageal Squamous Cell Carcinoma; 1.1 Acetaldehyde, a Metabolite of Alcohol, and the Development of Esophageal Squamous Cell Carcinoma; 1.2 Blood and Saliva Acetaldehyde Concentration After Alcohol Intake; 1.3 Acetaldehyde Reacts with DNA to Form DNA Adducts and Cause Severe DNA Damage; 1.4 Conclusions; References; Chapter 2: Gastroesophageal Reflux Diseases and Lifestyle Factors; 2.1 Introduction; 2.2 Obesity; 2.3 Metabolic Syndrome; 2.4 Diabetes Mellitus 2.5 Hypertension2.6 Dyslipidemia; 2.7 Cigarette Smoking; 2.8 Alcohol Drinking; 2.9 Late Meal; 2.10 Conclusions; References; Chapter 3: Post Helicobacter pylori Gastric Diseases; 3.1 H. pylori-Negative Mucosal Injury of the Stomach; 3.1.1 H. pylori-Negative Gastric Ulcer; 3.1.2 Drug-Induced Gastric Ulcer (Recurrent Gastric Ulcer) After H. pylori Eradication; 3.2 Inflammation (Gastritis) and Acid Secretion; 3.3 Functional Dyspepsia Defined without H. pylori Infection: Hypersensitive Mucosa to Acid Exposure; 3.4 Association of Non-alcoholic Fatty Liver Disease with Gastroesophageal Diseases 3.5 ConclusionsReferences; Chapter 4: New Perspectives in Gastric Cancer: Helicobacter pylori-Uninfected Pure Signet Ring Cell Carcinoma; 4.1 Introduction; 4.2 Diagnostic Criteria for HP-Uninfected Gastric Cancer; 4.3 Characteristics of HP-Uninfected Intramucosal Pure Signet Ring Cell Carcinoma; 4.3.1 Case Presentation of HDGC; 4.3.2 HP-Uninfected Pure Signet Ring Cell Carcinoma; 4.4 Speculation about the Carcinogenic Mechanisms of HP-Uninfected Pure Signet Ring Cell Carcinoma; 4.5 Future Directions; References; Chapter 5: Role of Alcohol and Metabolic Diseases in Colorectal Carcinogenesis 5.1 Colorectal Cancer Risk Attributable to Alcohol5.2 Risk for Precancerous Lesions in the Colorectum Attributable to Alcohol; 5.3 Developmental Mechanism of Colorectal Cancer Attributable to Alcohol; 5.4 Metabolic Syndrome and Colorectal Cancer Risk; 5.5 Colorectal Cancer Risk Attributable to Metabolic Syndrome; 5.6 Risk for Precancerous Lesions in the Colorectum Attributable to Metabolic Syndrome; 5.7 Four Components of Metabolic Syndrome and Colorectal Cancer Risk; 5.8 Developmental Mechanism of Colorectal Cancer Attributable to Metabolic Syndrome; 5.9 Epilogue; References Part II: Alcoholic/Non-Alcoholic Liver DiseasesChapter 6: Extracellular Vesicles in Alcoholic Liver Injury; 6.1 Introduction; 6.1.1 Alcoholic Liver Injury; 6.1.2 Extracellular Vesicles (EVs); 6.2 The Mechanism of EV Release and the Role of EVs in ALD/ASH; 6.2.1 Hepatocyte-Derived Extracellular Vesicles (Hep-EVs); 6.2.2 Non-parenchymal Cell-Derived Extracellular Vesicles; 6.3 EVs as Novel Biomarkers to Monitor Liver Injury in ALD/ASH; 6.3.1 Ligands and Proteins; 6.3.2 mtDNAs and miRNAs; 6.4 Conclusions; References; Chapter 7: Diabetes in Liver Disease
• Publisher Details: Singapore : Springer
• Publication Date: 2019
• Extent: 1 online resource
• Subjects: 616.3; Digestive organs -- Diseases; Electronic books; Electronic books
• Languages: English
• ISBNs: 9789811314650; 9811314659
• Related ISBNs: 9789811314643; 9811314640
• Notes: Note: Description based on online resource; title from digital title page (viewed on July 18, 2019).
• Access Rights: Legal Deposit; Only available on premises controlled by the deposit library and to one user at any one time; The Legal Deposit Libraries (Non-Print Works) Regulations (UK).
• Access Usage: Restricted: Printing from this resource is governed by The Legal Deposit Libraries (Non-Print Works) Regulations (UK) and UK copyright law currently in force.