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2. Calmodulin/CaMKII inhibition improves intercellular communication and impulse propagation in the heart and is antiarrhythmic under conditions when fibrosis is absent. Issue 4 (29th June 2016)

4. CTGF knockout does not affect cardiac hypertrophy and fibrosis formation upon chronic pressure overload. (November 2015)

5. Flotillins in the intercalated disc are potential modulators of cardiac excitability. (January 2019)

6. Multilevel analyses of SCN5A mutations in arrhythmogenic right ventricular dysplasia/cardiomyopathy suggest non-canonical mechanisms for disease pathogenesis. Issue 1 (9th January 2017)