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3. Endoglycan (PODXL2) is proteolytically processed by ADAM10 (a disintegrin and metalloprotease 10) and controls neurite branching in primary neurons. Issue 9 (14th August 2021)

4. Loss of TMEM106B potentiates lysosomal and FTLD‐like pathology in progranulin‐deficient mice. (14th September 2020)

5. The FTD‐like syndrome causing TREM2 T66M mutation impairs microglia function, brain perfusion, and glucose metabolism. (30th May 2017)