Search

Search Constraints

You searched for: Author/Creator Trozzi, L.

Search Results

1. Ageing-related expression of Twinfilin-1 regulates cholangiocyte biological response to injury. Issue 3 (October 2018)

2. Gut–liver axis derangement due to lack of inflammasome activity leads to visceral obesity and NASH development. (15th February 2015)

3. Identification of Twinfilin-1 as a key regulator of cholangiocyte biological response to injury: Evidence for a possible role of ageing in the progression of cholangiopathies. Issue 1 (16th February 2017)

4. Lack of NLRP3-inflammasome leads to gut-liver axis derangement and increases hepatic injury in a mouse model of NAFLD. (10th February 2016)

5. NLRP3 inflammasome increases hepatic fibrosis by inducing inflammatory signals in hepatic stellate cells. (15th February 2015)

6. OC.13.6: Identification of Twinfilin-1 as a Key Regulator of Cholangiocyte Biological Response to Injury: Evidence for a Possible Role of Ageing in the Progression of Cholangiopathies. (15th March 2017)

7. P.04.3 AGEING-RELATED EXPRESSION OF TWINFILIN-1 REGULATES CHOLANGIOCYTE BIOLOGICAL RESPONSE TO INJURY. Issue 2 (15th March 2018)

8. P.09.3 LACK OF NLRP3-INFLAMMASOME LEADS TO GUT-LIVER AXIS DERANGEMENT, GUT DYSBIOSIS AND A WORSENED PHENOTYPE IN A MOUSE MODEL OF NAFLD. Issue 2 (15th March 2018)

9. Selective LXRα intestinal activation reduces hepatic inflammation and fibrosis during the development of chronic liver injury. (15th February 2015)

10. The combination of mucus-degrading gram-negative bacteria and reduced antimicrobial peptides drives adipose tissue inflammation and NAFLD progression in mice lacking NLRP3-inflammasome. Issue 1 (16th February 2017)