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11. Ability of tau‐PET, phospho‐tau217, NfL and cortical thickness to predict short‐term cognitive decline in early symptomatic Alzheimer's disease. Issue 4 (1st February 2022)

12. Accurate risk estimation of β‐amyloid positivity to identify prodromal Alzheimer's disease: Cross‐validation study of practical algorithms. Issue 2 (23rd October 2018)

13. Acute phase markers in CSF reveal inflammatory changes in Alzheimer's disease that are impacted by APOE ε4, sex and age but not pathology: Biomarkers (non‐neuroimaging): Novel biomarkers. (7th December 2020)

14. Acute phase markers in CSF reveal inflammatory changes in Alzheimer's disease that intersect with pathology, APOE ε4, sex and age. (March 2021)

15. Age‐related tau‐PET uptake and its downstream effects extend beyond the medial temporal lobe in cognitively normal older adults. (20th December 2022)

16. Age‐related tau‐PET uptake and its downstream effects extend beyond the medial temporal lobe in cognitively normal older adults. (20th December 2022)

18. Amyloid‐associated increases in soluble tau is a key driver in accumulation of tau aggregates and cognitive decline in early Alzheimer. (20th December 2022)

19. Amyloid‐associated increases in soluble tau is a key driver in accumulation of tau aggregates and cognitive decline in early Alzheimer. (20th December 2022)