1. '(De-)sensitization' vs. 'Uncoupling': what drives cardiomyopathies in the thin filament? Reply. (13th October 2015) Authors: Marston, Steven; Messer, Andrew; Papadaki, Maria Journal: Cardiovascular research Issue: Volume 109:Number 1(2016) Page Start: 187 Record Type: Journal Article View Content: Available online (eLD content is only available in our Reading Rooms) ↗
2. Abnormal contractility in human heart myofibrils from patients with dilated cardiomyopathy due to mutations in TTN and contractile protein genes. Issue 1 (December 2017) Authors: Vikhorev, Petr; Smoktunowicz, Natalia; Munster, Alex; Copeland, O'Neal; Kostin, Sawa; Montgiraud, Cecile; Messer, Andrew; Toliat, Mohammad; Li, Amy; dos Remedios, Cristobal; Lal, Sean; Blair, Cheavar; Campbell, Kenneth; Guglin, Maya; Richter, Manfred; Knöll, Ralph; Marston, Steven Journal: Scientific reports Issue: Volume 7:Issue 1(2017) Page Start: 1 Record Type: Journal Article View Content: Available online (eLD content is only available in our Reading Rooms) ↗
3. Molecular defects in cardiac myofilament Ca2+- regulation due to cardiomyopathy-linked mutations can be reversed by small molecules binding to troponin. (November 2018) Authors: Sheehan, Alice; Messer, Andrew; Papadaki, Mary; Choudhry, Afnan; Křen, Vladimír; Biedermann, David; Blagg, Brian; Kandelwahl, Anuj Journal: Journal of molecular and cellular cardiology Issue: Volume 124(2018) Page Start: 87 Record Type: Journal Article View Content: Available online (eLD content is only available in our Reading Rooms) ↗