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2. Hyperactive HRAS dysregulates energetic metabolism in fibroblasts from patients with Costello syndrome via enhanced production of reactive oxidizing species. Issue 4 (11th September 2021)

3. IL-38 has an anti-inflammatory action in psoriasis and its expression correlates with disease severity and therapeutic response to anti-IL-17A treatment. Issue 11 (November 2018)

4. Paradoxical psoriasis induced by TNF‐α blockade shows immunological features typical of the early phase of psoriasis development. (29th October 2019)

5. Reduced concentrations of the B cell cytokine interleukin 38 are associated with cardiovascular disease risk in overweight subjects. Issue 3 (19th November 2020)

7. Selective Immunomodulation of Inflammatory Pathways in Keratinocytes by the Janus Kinase (JAK) Inhibitor Tofacitinib: Implications for the Employment of JAK-Targeting Drugs in Psoriasis. (19th November 2018)

8. Targeting CXCR4 by a selective peptide antagonist modulates tumor microenvironment and microglia reactivity in a human glioblastoma model. Issue 1 (December 2016)

9. The phosphoinositide-3-kinase (PI3K)-delta inhibitor seletalisib impairs monocyte-derived dendritic cells maturation, APC function, and promotes their migration to CCR7 and CXCR4 ligands. Issue 3 (24th February 2022)

10. The phosphoinositide‐3‐kinase (PI3K)‐delta inhibitor seletalisib impairs monocyte‐derived dendritic cells maturation, APC function, and promotes their migration to CCR7 and CXCR4 ligands. Issue 3 (24th February 2022)