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2. Cmah-dystrophin deficient mdx mice display an accelerated cardiac phenotype that is improved following peptide-PMO exon skipping treatment. (2nd October 2018)

3. Control of backbone chemistry and chirality boost oligonucleotide splice switching activity. Issue 10 (21st January 2022)

5. Uniform sarcolemmal dystrophin expression is required to prevent extracellular microRNA release and improve dystrophic pathology. Issue 2 (17th December 2019)