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3. Analysis of CLCNKB mutations at dimer‐interface, calcium‐binding site, and pore reveals a variety of functional alterations in ClC‐Kb channel leading to Bartter syndrome. Issue 4 (24th December 2019)

4. A new Kunitz‐type snake toxin family associated with an original mode of interaction with the vasopressin 2 receptor. (28th February 2022)

5. Brain renin-angiotensin system blockade with orally active aminopeptidase A inhibitor prevents cardiac dysfunction after myocardial infarction in mice. (February 2019)

6. Novel CLCNKB Mutations Causing Bartter Syndrome Affect Channel Surface Expression. Issue 9 (12th June 2013)

7. Emergence of Orai3 activity during cardiac hypertrophy. (11th September 2014)