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2. P4‐164: APOA‐I DEFICIENCY INCREASES CORTICAL AMYLOID DEPOSITION, CEREBRAL AMYLOID ANGIOPATHY, CORTICAL AND HIPPOCAMPAL ASTROGLIOSIS AND AMYLOID‐ASSOCIATED ASTROCYTE REACTIVITY IN APP/PS1 MICE. (1st July 2019)

3. O3‐01‐02: CHIMERA REPETITIVE MILD TRAUMATIC BRAIN INJURY INDUCES CHRONIC BEHAVIORAL AND NEUROPATHOLOGICAL PHENOTYPES IN WILD‐TYPE AND APP/PS1 MICE. (1st July 2019)

4. [P4–118]: HIGH‐DENSITY LIPOPROTEINS SUPPRESS Aβ‐INDUCED BRAIN MICROVASCULAR ENDOTHELIAL CELL ACTIVATION. (1st July 2017)

5. The effects of peripheral lipoprotein metabolism on cerebrovascular inflammation in APP/PS1 mice: Molecular and cell biology/vascular factors. (7th December 2020)

6. Cerebrovascular amyloid angiopathy in bioengineered vessels is reduced by high‐density lipoprotein particles enriched in apolipoprotein E: Development of new models and analysis methods/amyloid/Abeta. (7th December 2020)

7. [O2–04–01]: CLEARANCE OF BETA‐AMYLOID IS FACILITATED BY APOLIPOPROTEIN E AND CIRCULATING HIGH‐DENSITY LIPOPROTEINS IN BIOENGINEERED HUMAN VESSELS. (1st July 2017)

8. Microglial Cell Activation Increases Saturated and Decreases Monounsaturated Fatty Acid Content, but Both Lipid Species are Proinflammatory. Issue 4 (29th January 2014)